They found that a protein called bradykinin was produced in large amounts in the infected lungs. Bradykinin is a powerful signaling molecule which causes a drop in blood pressure and induces leakages in the blood vessels that allow both immune cells and fluid to escape from the blood and enter the interstitial spaces. In addition, they found overproduction of hyaluronic acid in the lung alveoli. Hyaluronic acid is a very long amino-sugar molecule to which heparan sulfate chains can attach to form gelled water. Through osmosis, hyaluronic acid will attract and immobilize the fluid escaping from the blood, and in this way induces pulmonary edema, a characteristic feature of ARDS (acute respiratory distress syndrome) . This can explain the sensation of drowning that COVID-19 patients have experienced. By trapping gelled water, the hyaluronic acid creates a negatively charged gel that releases protons into the interstitial spaces, to fuel the mitochondria of cells that are there.
4 Viral Lipid Envelope and Lipoxygenase
There is a class of enzymes called lipoxygenases that have an extraordinary ability to favor hydrogen over deuterium, and that produce therefore severely deuterium depleted water (DDW) by extracting hydrogen from fatty acids and combining it with oxygen . These enzymes depend on iron as a catalyst, but they are not flavoproteins, so they are not susceptible to disruption by glyphosate in the way that flavoproteins are. Lipoxygenase is upregulated during inflammatory conditions, and it oxidizes the lipids in the membranes of cells and in the LDL particles in the blood, modifying the lipids into molecules called leukotrienes that have powerful bioactive signaling capabilities. The cascade response to these leukotrienes leads to vascular leaks, edema and the accumulation of immune cells at the inflammatory site .
Remarkably, it has been discovered that the SARS CoV-2 virus protein coat contains three pockets within its contour shape that perfectly and specifically fit a very common fatty acid called linoleic acid . This is the most common fatty acid in the membranes of human cells, and it is surmised that the virus picks up multiple molecules of linoleic acid as it exits the human host cell, surrounding itself with a lipid envelope.
Since the immune cells respond to the virus by inducing an inflammatory response, and an inflammatory response induces lipoxygenase, it can be predicted that the linoleic acid trapped in the membranes of the viruses will be metabolized by lipoxygenase to produce leukotrienes, while also further supplying DDW to the surrounding fluids. This sets up a perfect situation for macrophages (“big eaters” – immune cells that specialize in clearing viruses) to congregate and partake of the sweet nectar that is being created through the immune response to the virus.
5 Mitochondrial Rejuvenation
It has only recently become known to researchers that cells have a remarkable ability to share their mitochondria among one another, and that this practice can lead to a healing process for sick immune cells.
We can now recognize that an acute reaction to SARS CoV-2 results in an intense inflammatory response that sets in motion a dramatic sequence of events, possibly with the ultimate magnanimous goal of energiziing the macrophages so that they can effectively clear the virus.
Such a response only happens when the immune cells are impaired due to prior toxic exposure, and I believe glyphosate is a major culprit. If the immune cells were healthy, they would have easily cleared the virus without overt symptoms of disease.