Masters of Health Magazine March 2020 | Page 10

2.Glyphosate shuts down the plants’ defense mechanism making it susceptible to disease causing organisms which are actually responsible for killing a susceptible plant that has been sprayed with a GBH. This has been demonstrated in several scientific studies by growing plants in sterile soil and then spraying them with a GBH. The plant will stop growing for up to 10 – 14 days and then begin to grow again after the growth hormones can function properly when the plant recovers/receives critic nutrients/minerals from the soil. The susceptible sprayed plant does NOT DIE but only stops growing.

3.This has also been demonstrated by applying a fungicide with the glyphosate herbicide. The disease(s) that kill plants that have been sprayed with glyphosate are promoted by the pathogenic fungi in the soil. The fungicide stops the fungal disease and thus prevents the herbicidal activity so the plants do not die. It has been stated for the ease of the lay person to better understand or make an association that “glyphosate gives the plant a bad case of AIDS”.

Again, it’s a defense mechanism in a plant (comparable to a human’s immune system) that is rendered inactive due to the presence of glyphosate.

4.Weeds are developing resistance to the diseases, not the glyphosate. This is just one reason, for the ever growing number of SUPER WEEDS.

Glyphosate

as an Antimicrobial Agent

First patented as an antimicrobial in 2004

1.Monsanto was granted a patent on glyphosate as an antibiotic (or parasite control product) in August 2004 and this patent was expanded in 2010. They applied for this patent in August 2003, concurrently while that the patent was being reviewed and approved, Monsanto denied that glyphosate could cause any possible damage to the soil or soil microorganisms.

Dr. Robert Kremer worked for the USDA ARS at Columbia, Missouri. He was unable for many years to get any of his detailed precise research published on the damaging effects of glyphosate to the soil, soil organisms and plants. This was due to Monsanto’s influence in the scientific journal arena in the United States. Dr. Kremer submitted his research to the European Journal of Agronomy to finally get the work published. The most interesting part about glyphosate’s antimicrobial effect is that it affects beneficial organisms at much lower doses than it does the opportunistic or pathogenic disease-causing organisms.

This has been documented in soil by Dr. Kremer & others, in livestock; chickens by Dr. Monika Krueger as well as causing botulism in cattle. It bears repeating that glyphosate has been proven to stimulate the growth of the opportunistic/pathogenic disease-causing organisms at very low levels. A consequence of this differential toxicity reduces the population of the biological control organisms (the good bugs) and increases the population of the opportunistic or disease causing organisms (the bad bugs). Fusarium is an example of a disease causing organism.

Glyphosate also causes a continual increased veracity or intensity of the opportunistic organisms. So, the bad bugs continue to maintain or increase their population (throughout the growing season) and keep getting stronger wherever/whenever glyphosate is present in the environment.